Mar 18 β€’ 22:25 UTC πŸ‡°πŸ‡· Korea Hankyoreh (KR)

The concentration of hepatitis virus, not liver enzymes, is more effective in preventing liver cancer

A new perspective suggests that the concentration of hepatitis virus rather than liver enzyme levels is more indicative of liver cancer prevention.

In a recent discussion, new insights into liver health have emerged, particularly regarding the relevance of liver enzyme levels versus hepatitis virus concentration. Professor Lim Young-seok, the new president of the Korean Association for the Study of the Liver, explains that liver enzymes often mislead health assessments, leading to unnecessary anxiety among patients. The discussion highlights the importance of understanding that while elevated enzyme levels can indicate liver issues, they do not always correlate with the existence of hepatitis virus, which poses a more significant risk factor for conditions like liver cirrhosis and liver cancer.

The professor uses an analogy comparing the liver to a large factory, where liver cells produce necessary substances for the body. Conditions like fatty liver, hepatitis, and cirrhosis are likened to fires occurring within this factory. The hepatitis B virus is identified as a potential spark that can ignite such fires, resulting in 70-75% of cirrhosis cases and 60% of liver cancer cases in South Korea being attributed to hepatitis B. In contrast, hepatitis B antibodies act as firefighters that prevent or extinguish these potential fires, underlining the need for effective monitoring of virus concentration in patients rather than simply focusing on enzyme levels.

The discussion reiterates that the presence of hepatitis B virus without accompanying antibodies indicates a high risk and necessitates proactive intervention, while positive antibodies without virus indicate a controlled and non-threatening status. This streamlined understanding aims to empower patients in interpreting their health tests accurately, emphasizing preventive measures against liver diseases caused by hepatitis rather than over-focusing on transient enzyme fluctuations.

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